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In patients with T2DM and established CV disease, empagliflozin and canagliflozin have been shown to reduce the of MACE events, although only empagliflozin has demonstrated an ability to reduce the risk of CV death in this population

Indeed, a number of mitochondrial abnormalities are seen in both disease-affected individuals and animal models, including reduced calcium buffering capability, compromised bioenergetics, increased oxidative stress, and aberrant trafficking and dynamics [47]

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