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Description

It acts as a key transporter of long-chain fatty acids into mitochondria, where these fats are oxidized to produce adenosine triphosphate (ATP), the cells energy currency

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This stability supports its use across biochemical, cellular and preclinical experimental platforms

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Bone formation induced by intermittent PTH administration to patients with osteoporosis could be explained, at least in part, by the ability of PTH to downregulate sclerostin expression in osteocytes, permitting the anabolic Wnt signaling pathway to proceed (230)

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