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Catalpol alleviates isoflurane-induced hippocampal learning and memory dysfunction and neuropathological changes in aged mice

Aos, upon binding to cell-surface receptors on neurons, disrupt signal transduction, leading to a sustained interruption of intracellular Ca 2+ signaling through increased N-methyl-D-aspartate (NMDA) receptor-dependent Ca 2+ influx [77]

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This is especially important in the context of obesity treatment, where inappropriate prescribing practices have been a concern