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In summary, PCOS patients could be classified into phenotype A, B, C, or D based on manifest symptoms (Table 1) [46]

Importantly, these implications were observed without direct immunosuppression of specific cytokines such as TNF, implying a more regulatory mode of action

Assessment of severity is particularly relevant to decide on whether a given patient should be treated by aggressive treatments (either medical or surgical) or simply by local or general supportive measures (see below).The other important feature of GO is its activity

This suggests that butyrate modulates gene expression rather than receptor stimulation [37]