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In response to oxidative stress, neurons undergo overactivation of the enzyme poly (ADP-ribose) polymerase-1 (PARP1) to prevent DNA damage, which decreases intracellular NAD and ATP levels, compromising the efficiency of glycolysis and oxidative phosphorylation, affecting the ability of neurons to generate ATP (200, 203)

This powerful amplification loop then drastically activates the complement system, leading to massive release of inflammatory factors, formation of membrane attack complexes, and cell lysis, all of which significantly exacerbate brain tissue damage

Taking glutathione with food or reducing the initial dose often helps minimize these effects