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doi: 10.1136/gutjnl-2021-324243 63

To maintain iron homeostasis, cells employ intricate regulatory mechanisms, including iron uptake via the transferrin receptor (TFR1), storage of iron in ferritin, and ferritinophagy to mitigate the toxic effects of iron overload ( In addition to these well-characterized mechanisms, recent studies have highlighted alternative ferroptosis surveillance pathways that operate independently of GPX4

The mechanistic logic holds: GHK-Cu operates largely via transcriptional gene regulation (slower, 4 to 8 weeks for measurable effect) while TB-500 operates via cytoskeletal and cytokine pathways (faster, 1 to 3 weeks)