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Brigelius-Flohe R, Maiorino M

through exercise) coupled with rest and glutathione administration can optimize glutathione levels in the joints and delay the aging of cartilage [7]

However, more recent studies assessing the role of ferrous iron in the pathophysiology first in vitro [74, 75] and then in vivo [76, 77] provided evidence that after a toxic dose of APAP ferrous iron derived from lysosomes is taken up into mitochondria through a Ca 2+ uniporter and facilitates the MPTP opening and cell death

Int J Mol Med 33:687694 Funding This work was supported by the National Natural Science Foundation of China [No.82000920] and the special fund for medical talents of Jilin Province, China [2019SRCJ022]