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Consistent with our data identifying different regulation of incretin bioactivity and modulation of glucose metabolism with DPP4 in hepatocytes versus Tie2 + cells, recent reports have determined that elevated levels of active portal GLP-1 are disconnected from the classic definition of the incretin effect as increased portal circulation of GLP-1 does not potentiate nutrient-stimulated insulin secretion (64, 65)
Subsequently, several studies also revealed other effects, such as intensifying cell motility, angiogenesis, immune response, cell differentiation, and anti-apoptosis (Garcia-Vilas and Medina, 2018)
10.1007/s12975-010-0063-z Transl
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