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Accordingly, acute carnitine deficiency induced by excessive conversion of free carnitine to acetylcarnitine could lead to detrimental changes in mitochondrial functionality during ischemia and intensive exercise

To this point, a review of 48 trials demonstrated that carnitine supplementation significantly lowered various markers of inflammation like C-reactive protein, interleukin 6 (IL-6), and tumor necrosis factor (TNF), while increasing total antioxidant capacity (8)

Further research on the molecular mechanisms of these supplements will enhance their clinical application by elucidating their role in mitigating oxidative stress and optimizing mitochondrial function

MicroRNAs: Small RNAs with a big role in gene regulation (vol 5, pg 522 2004)