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It was originally developed as a potential anti-obesity drug due to its ability to influence fat metabolism without causing the same growth-promoting effects associated with full HGH therapy (review)
At each time point, 1/10 volume of culture medium per well CCK-8 solution reagent was added, incubated at 37C for 3 h, and the absorbance was measured at 450 nm using a microplate reader
Attenuated mitochondrial permeability transition and maintained nearly 100% survival at 48 hours under high-dose lipopolysaccharide challenge
At the cellular level, hypoxia disrupts mitochondrial energy metabolism by shifting ATP production from oxidative phosphorylation to anaerobic glycolysisa transition mediated by hypoxia-inducible factor-1 (HIF-1
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