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Abstract Current gold standard therapeutic strategies for T2DM target insulin resistance or cell dysfunction as their core mechanisms of action

MEK and TAK1 contain the conserved cysteine residue corresponding to ERK2 (95)

Interestingly, MTX + mildronate samples (group 5) showed more severe tubular damage and degenerative changes (red arrow) accompanied with hyperplasia of mesangial cells of renal corpuscles (black arrow head), mild interstitial inflammatory cells infiltrates were showed (red arrow head) with obvious higher fibroblastic activity as well as periglomerular and interstitial fibrosis (dashed arrow)

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