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Under the control condition, the PIN2-GFP signal was preferentially distributed to the apical side of the epidermal cells, and the internalized PIN2-GFP signal was also frequently observed (Fig

CETP mice injected with either vehicle, a GIPR agonist (GIPFA-085), a GLP1R agonist (GLP-140) or both agonists presented similar results ( and proatherogenic peptides and systemic inflammation markers ICAM-1 and VCAM-1 involved in monocytes/macrophages adhesion to endothelium