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Description

[12] Damage to the liver is not due to the drug itself but to a toxic metabolite ( N -acetyl- p -benzoquinone imine (NAPQI)) produced by cytochrome P-450 enzymes in the liver

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GSH is primarily produced in liver cells and cells throughout the central nervous system, although it is also present in the foods we eat every day, such as fish, poultry, and fruits

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Together, these actions constitute the molecular mechanisms underlying TMAOs pro-thrombotic effects [42]

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