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By acting as a hepatocyte growth factor (HGF)/c-Met agonist, it promotes neuroplasticity, memory formation, and potential repair of damaged neural circuits
GHK-Cus documented NGF stimulation appears to sit within its broader gene expression modulation signature rather than representing a direct receptor interaction, which is mechanistically coherent with the transcriptomic reactivation data showing GHK-Cu shifting aging gene expression profiles toward younger patterns (PMID: 25904764)
doi: 10.1016/j.bbrc.2018.09.062 73 GeorgeSLuceroYTorresJPLagomarcinoAJORyanM
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